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Hans‐Peter Zenner

机构信息待补充

43H指数
2.7K论文数
7.1K被引数
收录论文 15
发表时间
Substitutions in the conserved C2C domain of otoferlin cause DFNB9, a form of nonsyndromic autosomal recessive deafness
err2002-07-01
err56
PREAI
errMirghomizadeh, F; Pfister, M; Apaydin, F; Petit, C; Kupka, S; Pusch, CM; Zenner, HP; Blin, N
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Neonatal rat cartilage has the capacity for tissue regeneration
err2002-03-12
err15
PREAI
errWagner, W; Reichl, J; Wehrmann, M; Zenner, HP
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Thyroid hormone-deficient period prior to the onset of hearing is associated with reduced levels of β-tectorin protein in the tectorial membrane -: Implication for hearing loss
err2001-10-01
err64
errOAAI
errKnipper, M; Richardson, G; Mack, A; Müller, M; Goodyear, R; Limberger, A; Rohbock, K; Köpschall, I; Zenner, HP; Zimmermann, U
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Three-dimensional motion of the organ of Corti
err2000-05-01
err70
errOAAI
errHemmert, W; Zenner, HP; Gummer, AW
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Molecular characterization of anion exchangers in the cochlea
err2000-01-01
err10
PREAI
errZimmermann, U; Köpschall, I; Rohbock, K; Bosman, GJCGM; Zenner, HP; Knipper, M
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Gene disruption of p27Kip1 allows cell proliferation in the postnatal and adult organ of Corti
err1999-03-30
err344
errOAAI
errLöwenheim, H; Furness, DN; Ki, J; Zinn, C; Gültig, K; Fero, ML; Frost, D; Gummer, AW; Roberts, JM; Rubel, EW; Hackney, CM; Zenner, HP
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Desensitization of the P2X(2) receptor controlled by alternative splicing
err1997-11-07
err154
errOAAI
errBrandle, U; Spielmanns, P; Osteroth, R; Sim, J; Surprenant, A; Buell, G; Ruppersberg, JP; Plinkert, PK; Zenner, HP; Glowatzki, E
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Identification of a titratable lysine residue that determines sensitivity of kidney potassium channels (ROMK) to intracellular pH
err1996-08-15
err172
errOAAI
errFakler, B; Schultz, JH; Yang, J; Schulte, U; Brandle, U; Zenner, HP; Jan, LY; Ruppersberg, JP
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Extracellular K+ and intracellular pH allosterically regulate renal K(ir)1.1 channels
err1996-07-01
err71
PREAI
errDoi, T; Fakler, B; Schultz, JH; Schulte, U; Brandle, U; Weidemann, S; Zenner, HP; Lang, F; Ruppersberg, JP
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K(IR)2.1 INWARD RECTIFIER K+ CHANNELS ARE REGULATED INDEPENDENTLY BY PROTEIN-KINASES AND ATP HYDROLYSIS
errNEURON
IF15
err1994-12-01
err151
PREAI
errFAKLER, B; BRANDLE, U; GLOWATZKI, E; ZENNER, HP; RUPPERSBERG, JP
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