Zhong-Xiuzi Gao

nephrology

H指数:20

被引数:1,103

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Emerging multidimensional biomarker system for cardiovascular-kidney-metabolic syndrome: from multi-omics integration to clinical artificial intelligence

心血管-肾脏-代谢综合征的新兴多维生物标志物系统: 从多组学整合到临床人工智能

Cardiovascular Diabetology

IF10.6

2026-04-260

Lack of Kir4.1 in the Distal Convoluted Tubule Causes ENaC Hyperactivity During K+ Restriction Leading to Hypokalemia

远曲小管缺乏Kir4.1导致K⁺限制期间ENaC过度活跃并引发低钾血症

Acta Physiologica

IF5.6

2026-04-140

PRE

Macrophage-driven inflammation in acute kidney injury: Therapeutic opportunities and challenges

急性肾损伤中巨噬细胞驱动的炎症: 治疗的机遇和挑战

TRANSLATIONAL RESEARCH

IF5.9

2025-04-010

PRE

Soluble (pro)renin receptor as a novel regulator of renal medullary Na+ reabsorption

AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY

IF3.4

2025-02-010

PRE

Impaired distal renal potassium handling in streptozotocin-induced diabetic mice

链脲佐菌素诱导的糖尿病小鼠远端肾钾处理受损

AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY

IF3.4

2024-07-011

PRE

Defective natriuresis contributes to hyperkalemia in db/db mice during potassium supplementation

JOURNAL OF HYPERTENSION

IF4.1

2024-05-201

PRE

Ferroptosis: a potential bridge linking gut microbiota and chronic kidney disease

CELL DEATH DISCOVERY

IF7

2024-05-155

OAAI

Kir4.1 deletion prevents salt-sensitive hypertension in early streptozotocin-induced diabetic mice via Na+-Cl- cotransporter in the distal convoluted tubule

Kir4.1缺失通过远端回旋小管中的na-cl-共转运蛋白预防早期链脲佐菌素诱导的糖尿病小鼠的盐敏感性高血压

JOURNAL OF HYPERTENSION

IF4.1

2023-04-053

PRE

Activation of Kir4.1/Kir5.1 contributes to the cyclosporin A-induced stimulation of the renal NaCl cotransporter and hyperkalemic hypertension

Kir4.1/Kir5.1的激活有助于环孢菌素A诱导的肾脏NaCl协同转运蛋白的刺激和高血钙性高血压

ACTA PHYSIOLOGICA

IF5.6

2023-02-167

PRE

Gut microbiota and its metabolites - molecular mechanisms and management strategies in diabetic kidney disease

FRONTIERS IN IMMUNOLOGY

IF5.9

2023-01-1919

OAAI

Identification of ULK1 as a novel mitophagy-related gene in diabetic nephropathy

FRONTIERS IN ENDOCRINOLOGY

IF4.6

2023-01-1814

OAAI

Single-cell transcriptomics: A new tool for studying diabetic kidney disease

FRONTIERS IN PHYSIOLOGY

IF3.4

2023-01-048

OAAI

Deletion of Kir5.1 abolishes the effect of high Na+ intake on Kir4.1 and Na+-Cl- cotransporter

Kir5.1的缺失消除了高Na摄入对Kir4.1和na-cl-协同转运蛋白的影响

AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY

IF3.4

2021-06-0113

Deletion of renal Nedd4-2 abolishes the effect of high sodium intake (HS) on Kir4.1, ENaC, and NCC and causes hypokalemia during high HS

AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY

IF3.4

2021-05-0118

Deletion of NEDD4-2 Abolishes the Inhibitory Effect of High Sodium (HS) Intake on the Basolateral Kir4.1/Kir5.1 of the DCT and Thiazide-Sensitive Na-Cl Cotransport (NCC)

FASEB JOURNAL

IF4.2

2020-04-160

Renal Tubule Nedd4-2 Deficiency Stimulates Kir4.1/Kir5.1 and Thiazide-Sensitive NaCl Cotransporter in Distal Convoluted Tubule

JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

IF9.4

2020-04-1518

OAAI

Effect of Angiotensin II on ENaC in the Distal Convoluted Tubule and in the Cortical Collecting Duct of Mineralocorticoid Receptor Deficient Mice

血管紧张素II对盐皮质激素受体缺陷小鼠远曲小管和皮质集合管中ENaC的影响

JOURNAL OF THE AMERICAN HEART ASSOCIATION

IF5.3

2020-04-0941

OAAI

Deletion of Kir5.1 Impairs Renal Ability to Excrete Potassium during Increased Dietary Potassium Intake

JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

IF9.4

2019-06-2543

OAAI

Norepinephrine-Induced Stimulation of Kir4.1/Kir5.1 Is Required for the Activation of NaCl Transporter in Distal Convoluted Tubule

HYPERTENSION

IF8.2

2019-01-0122

OAAI

Kir4.1/Kir5.1 Activity Is Essential for Dietary Sodium Intake-Induced Modulation of Na-Cl Cotransporter

JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

IF9.4

2018-12-1735

OAAI

Zhong-Xiuzi Gao

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