Zoledronic Acid Blocks Overactive Kir6.1/SUR2-Dependent KATP Channels in Skeletal Muscle and Osteoblasts in a Murine Model of Cantu SyndromeCELLSIF5.22023-03-173OAAI
Consequences of SUR2[A478V] Mutation in Skeletal Muscle of Murine Model of Cantu SyndromeCELLSIF5.22021-07-1512OAAI
Complex consequences of Cantu syndrome SUR2 variant R1154Q in genetically modified miceSJCI INSIGHTIF6.12021-03-0813OA
The Mechanism of High-Output Cardiac Hypertrophy Arising From Potassium Channel Gain-of-Function in Cantu SyndromeFUNCTIONIF3.82020-06-1819OAAI
Glibenclamide reverses cardiovascular abnormalities of Cantu syndrome driven by KATP channel overactivity ConorJOURNAL OF CLINICAL INVESTIGATIONIF13.62020-02-1737OA
ABCC9-related Intellectual disability Myopathy Syndrome is a KATP channelopathy with loss-of-function mutations in ABCC9NATURE COMMUNICATIONSIF15.72019-10-0137OAAI
The Mechanism of Cardiovascular Pathophysiology in Cantu Syndrome and Response to Glibenclamide in Novel KATP Channel Mutant Mouse ModelsBIOPHYSICAL JOURNALIF3.12019-02-010OAAI
Cardiovascular Consequences of Cantu Syndrome and Response to Glibenclamide Treatment in Two Novel KATP Channel Mutant Mouse ModelsARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGYIF7.42018-05-010PRE
Hypotension Due to Kir6.1 Gain-of-Function in Vascular Smooth MuscleJOURNAL OF THE AMERICAN HEART ASSOCIATIONIF5.32013-08-2262OAAI
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