arrow
F

Francesco Paolo Busardò

Marche Polytechnic University
H指数:47
被引数:7,626

个人简介

暂无内容

教育背景

暂无内容

工作经历

暂无内容

荣誉奖项

暂无内容

Toxicokinetics and analytical toxicology of four N, N-dimethyltryptamine derivatives studied in human in vitro systems and in vivo by means of zebrafish embryos

四种N,N-二甲基色胺衍生物的毒代动力学与分析毒理学研究:体外人源系统和斑马鱼胚胎体内实验

2026-09-210
OAAI
Simultaneous enantioselective separation of 2-, 3-and 4-chloromethcathi-nones using supercritical fluid chromatography-tandem mass spectrometry and its application to human oral fluid samples

超临界流体色谱-串联质谱法同时手性分离2-、3-和4-氯甲硫丙酮及其在人唾液样本中的应用

2026-06-010
PRE
Enantioselective determination of methylone and its major phase-1 metabolites in various biological matrices and stereochemical stability of methylone enantiomers

甲基酮及其主要I相代谢物在不同生物基质中的立体选择性测定和甲基酮对映体的立体化学稳定性

2026-05-200
PRE
5-Methyl etodesnitazene human metabolism: LC-ESI±-HRMS/MS analysis (mono- and di-protonation) of human hepatocyte incubations and positive biospecimens

5-甲基乙氧基地氮西因的人体代谢:人体肝细胞孵育和阳性生物样本的LC-ESI±-HRMS/MS分析(单-和双-质子化)

2026-04-280
OAAI
Human metabolism and pharmacological profiling of protonitazepyne and metonitazepyne, two highly potent nitazenes: prediction of main metabolite activity based on µ-opioid receptor docking simulations

人代谢和药理学特征分析:两种高度活性的硝卓类物质protonitazepyne和metonitazepyne——基于μ-阿片受体对接模拟的主要代谢物活性预测

2025-10-310
OA
Synthetic cannabinoid receptor agonists containing silicon: exploring the metabolic pathways of ADMB- and Cumyl-3TMS-PrINACA in human urine specimens and post mortem material compared to in vitro and in silico data

含硅合成大麻素受体激动剂:探索ADMB-和Cumyl-3TMS-PrINACA在人体尿液样本和死后材料中的代谢途径,并与体外和计算机模拟数据比较

2025-10-070
OA
In vivo and in vitro metabolism of the designer benzodiazepine, bretazenil: a comparison of pooled human hepatocytes and liver microsomes with postmortem urine and blood samples

体内和体外代谢研究:以设计型苯二氮䓬类药物bretazenil为例——比较混合人肝细胞和肝微粒体与死后尿液和血液样本

2025-10-010
OAAI
Molecular and Computational Studies Reveal That Per- and Polyfluoroalkyl Substances Can Impair Protamine–DNA Interaction, Potentially Inducing DNA Damage

分子和计算研究揭示,全氟和多氟烷基物质可以损害原精蛋白–DNA相互作用,可能诱导DNA损伤

2025-09-040
OAAI