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Herrock, Owen

university of gothenburg

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Disrupted brain angiogenesis and blood–brain barrier function underlie cognitive deficits in offspring of preeclampsia-like pregnancies

The Journal of Physiology

IF0

2026-01-280

PRE

Exenatide Is Neuroprotective in a New Rabbit Model of Hypoxia-Ischemia

Exenatide 在新型缺氧-缺血兔模型中具有神经保护作用

Cells 2025, Vol. 14, Page 1715

IF5.2

2025-11-010

OAAI

Adoptive transfer of placental CD4+T Cells from preeclamptic patients with a history of COVID-19 causes hypertension and cognitive dysfunction postpartum in a pregnant rat model of preeclampsia

IF8.2

2024-09-010

PRE

AT1-AA Is Produced in Offspring in Response to Placental Ischemia and Is Lowered by B-Cell Depletion Without Compromising Overall Offspring Health

JOURNAL OF THE AMERICAN HEART ASSOCIATION

IF5.3

2024-02-200

OAAI

Tnf-α Blockade During Pregnancy Does Not Impact Offspring Development In A Rat Model Of Preeclampsia

IF8.2

2023-09-010

PRE

Diabetic CD4+T Cells Causes Increased Glucose, Hypertension, Renal Injury, And Decreased Vascular Endothelial Cell Function In A Novel Rat Model Of Diabetes During Pregnancy

IF8.2

2023-09-010

PRE

CD4+T Cells From Preeclamptic Patients During Pregnancy Causes Cognitive Dysfunction Postpartum In A Pregnant Rat Model

IF8.2

2023-09-010

PRE

Inhibition of angiotensin II type 1 receptor agonistic autoantibodies by direct binding does not impact reduced uterine perfusion pressure offspring birthweight and blood pressure at adulthood

AMERICAN JOURNAL OF OBSTETRICS & GYNECOLOGY MFM

IF3.1

2023-06-012

OAAI

Setting a stage: Inflammation during preeclampsia and postpartum

FRONTIERS IN PHYSIOLOGY

IF3.4

2023-02-238

OAAI

B2 cells contribute to hypertension and natural killer cell activation possibly via AT1-AA in response to placental ischemia

AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY

IF3.4

2023-02-018

OA

The role of immune cells and mediators in preeclampsia

NATURE REVIEWS NEPHROLOGY

IF39.8

2023-01-1267

OA

The Role of Different Lymphoid Cell Populations in Preeclampsia Pathophysiology

IF3

2022-10-274

OA

Gestational Diabetic Cd4+T Cells Cause Increased Glucose, Blood Pressure And Placental Mitochondrial Dysfunction In A Novel Rat Model Of Diabetes During Pregnancy

IF8.2

2022-09-010

PRE

B Cells Induce Hypertension, AT1AA, And Complement Activation In Preeclampsia Or In Response To A Prior Covid-19 Infection During Pregnancy

IF8.2

2022-09-010

PRE

CD4+T Cells cause increased glucose, mitochondrial dysfunction, and hypertension in a Novel Pregnant Rodent Model of Gestational Diabetes Mellitus

IF4.2

2022-05-130

PRE

The Role of B Cells in Mediating Hypertension in Preeclampsia or COVID-19 Infection During Pregnancy

IF4.2

2022-05-133

OA

Progesterone prolongs time to delivery and attenuates blood pressure possibly by improving inflammation and endothelial function in response to preeclampsia

IF4.2

2022-05-130

PRE

IL17 administration in the Absence of T cells Results in Hypertension, NK cell Activation, and Reduced Pup Weight at Birth, but No Changes in Blood Pressure or Weight at Maturation of Offspring

IF4.2

2022-05-130

PRE

Maternal B Cell Depletion Reduces Blood Pressure and Improves Fetal Weights in Male Offspring of a Rat Model of Preeclampsia

IF4.2

2022-05-130

PRE

Progesterone Induced Blocking Factor Reduces Hypertension and Placental Mitochondrial Dysfunction in Response to sFlt-1 during Pregnancy

IF5.2

2021-10-2014

OAAI