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Sarah Fitzgerald

university of mississippi

H指数:9

被引数:453

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B2 cells contribute to hypertension and natural killer cell activation possibly via AT1-AA in response to placental ischemia

AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY

IF3.4

2023-02-018

OA

The role of immune cells and mediators in preeclampsia

NATURE REVIEWS NEPHROLOGY

IF39.8

2023-01-1267

OA

Gestational Diabetic Cd4+T Cells Cause Increased Glucose, Blood Pressure And Placental Mitochondrial Dysfunction In A Novel Rat Model Of Diabetes During Pregnancy

IF8.2

2022-09-010

PRE

B Cells Induce Hypertension, AT1AA, And Complement Activation In Preeclampsia Or In Response To A Prior Covid-19 Infection During Pregnancy

IF8.2

2022-09-010

PRE

CD4+T Cells cause increased glucose, mitochondrial dysfunction, and hypertension in a Novel Pregnant Rodent Model of Gestational Diabetes Mellitus

IF4.2

2022-05-130

PRE

IL17 administration in the Absence of T cells Results in Hypertension, NK cell Activation, and Reduced Pup Weight at Birth, but No Changes in Blood Pressure or Weight at Maturation of Offspring

IF4.2

2022-05-130

PRE

Maternal B Cell Depletion Reduces Blood Pressure and Improves Fetal Weights in Male Offspring of a Rat Model of Preeclampsia

IF4.2

2022-05-130

PRE

Progesterone Induced Blocking Factor Reduces Hypertension and Placental Mitochondrial Dysfunction in Response to sFlt-1 during Pregnancy

IF5.2

2021-10-2014

OAAI

Low Dose of IL-2 Normalizes Hypertension and Mitochondrial Function in the RUPP Rat Model of Placental Ischemia

IF5.2

2021-10-196

OAAI

IL-17 Causes Hypertension, Reduced Fetal Weight And Natural Killer Cell Activation In The Absence Of T Cells

IF8.2

2021-09-010

PRE

Maternal B Cell Depletion Lowers Blood Pressure And Improves Fetal Weights In Offspring In A Rat Model Of Preeclampsia

IF8.2

2021-09-010

PRE

Cd4+t Cells Cause Hypertension, Increased Glucose, And Mitochondrial Dysfunction In A Novel Rodent Model Of Gestational Diabetes Mellitus

IF8.2

2021-09-010

PRE

Inhibiting B Cell Activating Factor Normalizes The Hypertension And Cytolytic Natural Killer Cells Associated With Placental Ischemia

IF8.2

2021-09-010

PRE

IL-17 causes hypertension and multi-organ tissue dysfunction which is attenuated with blockade of agonistic autoantibodies to the angiotensin II type I (AT1-AA) receptor during pregnancy

IF4.2

2021-05-140

PRE

CD4+T cells cause renal and placental mitochondrial oxidative stress as mechanisms of hypertension in response to placental ischemia

CD4 T细胞引起肾和胎盘线粒体氧化应激作为高血压响应胎盘缺血的机制

AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY

IF3.4

2021-01-0125

OAAI

Inhibition Of T Cell Activation In Response To Placental Ischemia During Pregnancy Improves Hypertension And Activation Of Natural Killer Cells

IF8.2

2020-09-010

PRE

Rupp Rat Model Cd4+T Cells Activate Nk Cells And Cause Mitochondrial Oxidative Stress And Hypertension In Normal Pregnant Rats

IF8.2

2020-09-010

PRE

Mitochondrial Dysfunction And Natural Killer Cell Activation Stimulated By Il-17 Signaling From Th17 Cells In Response To Placental Ischemia During Pregnancy

IF8.2

2020-09-010

PRE

B Cells Contribute To Hypertension And Cytolytic Nk Cell Activation In Response To Placental Ischemia In Pregnancy

IF8.2

2020-09-010

PRE

Comparison of the formation, adipogenic maturation, and retention of human adipose-derived stem cell spheroids in scaffold-free culture techniques

JOURNAL OF BIOMEDICAL MATERIALS RESEARCH PART B-APPLIED BIOMATERIALS

IF3.4

2020-05-1213

OAAI