Chitra Thakur

stony brook cancer center

H指数:21

被引数:1,325

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Utilization Rates and Factors Associated with Adoption of Complementary and Alternative Medicine Practices in the US

美国补充与替代医学实践的利用率及其影响因素

Journal of Evidence-Based Integrative Medicine

IF3.6

2026-07-090

PRE

Histone code rewriting in mdig-deficient triple-negative breast cancer cells drives metastasis-promoting transcription

组蛋白密码重写通过在mdig缺陷的三阴性乳腺癌细胞中驱动促转移转录

Genes & Diseases

IF9.4

2026-03-040

OAAI

Lung cancer in women: current evidence and future research priorities

女性肺癌：当前证据和未来研究重点

Lung Cancer

IF4.4

2026-01-040

PRE

Nrf2 Drives Epigenetic Reprogramming and Acts as the Master Regulator of KLF4 Expression and Activity in Arsenic-Induced Transformation

Nrf2驱动表观遗传重编程，并作为KLF4表达和活性的主调节因子，在砷诱导的转化过程中发挥作用。

Advanced Science

IF14.1

2025-08-040

OAAI

Genome-wide mapping of arsenic-activated Nrf2 reveals metabolic and epigenetic reprogramming in induced pluripotent stem cells

全基因组绘制砷激活的Nrf2，揭示了诱导多能干细胞中的代谢和表观遗传重编程

Redox Biology

IF11.9

2025-07-170

PREAI

1,4-Dioxane Induces Epithelial-Mesenchymal Transition and Carcinogenesis in an Nrf2-Dependent Manner

1,4-二氧六环以Nrf2依赖性方式诱导上皮-间质转化和致癌作用

JOURNAL OF EXTRACELLULAR VESICLES

IF14.5

2025-04-300

OAAI

Aryl Hydrocarbon Receptor (AHR) Suppresses Arsenic (As3+)-Induced Malignant Transformation by Antagonizing TOX Expression

芳基羟化物受体（AHR）通过拮抗TOX表达抑制砷（As3+）诱导的恶性转化

INTERNATIONAL JOURNAL OF BIOLOGICAL SCIENCES

IF10

2025-03-310

Nrf2 and HIF1α converge to arsenic-induced metabolic reprogramming and the formation of the cancer stem-like cells (vol 10, pg 4134, 2020)

Nrf2和HIF1α在砷诱导的代谢重编程和癌干细胞样细胞的形成中汇聚（卷10，页4134，2020）

THERANOSTICS

IF13.3

2025-03-150

Editorial: The impact of specific environmental exposures on breast, lung, and colon cancer: advancing public health strategies for enhanced outcomes

FRONTIERS IN PUBLIC HEALTH

IF3.4

2025-02-140

OAAI

Genomic and epigenetic characterization of the arsenic-induced oncogenic microRNA-21

ENVIRONMENTAL POLLUTION

IF7.3

2024-03-010

OAAI

Tumor suppressive activity of AHR in environmental arsenic-induced carcinogenesis

TOXICOLOGY AND APPLIED PHARMACOLOGY

IF3.4

2023-12-012

OAAI

Epigenetic regulation of breast cancer metastasis

乳腺癌转移的表观遗传调控

CANCER AND METASTASIS REVIEWS

IF8.7

2023-10-194

PRE

Epigenetics and environment in breast cancer: New paradigms for anti-cancer therapies

乳腺癌的表观遗传学和环境: 抗癌治疗的新范式

FRONTIERS IN ONCOLOGY

IF3.3

2022-09-1526

OAAI

Profiling of histone H3 trimethylation and distinct epigenetic pattern of chromosome Y in the transformed bronchial epithelial cells induced by consecutive arsenic treatment

GENES & DISEASES

IF9.4

2022-09-015

OAAI

Depletion of Mdig Changes Proteomic Profiling in Triple Negative Breast Cancer Cells

BIOMEDICINES

IF3.9

2022-08-190

OAAI

Arsenic Activates the ER Stress-Associated Unfolded Protein Response via the Activating Transcription Factor 6 in Human Bronchial Epithelial Cells

BIOMEDICINES

IF3.9

2022-04-224

OAAI

Arsenic activates STAT3 signaling during the transformation of the human bronchial epithelial cells

TOXICOLOGY AND APPLIED PHARMACOLOGY

IF3.4

2022-02-017

OAAI

Cooperation between NRF2-mediated transcription and MDIG-dependent epigenetic modifications in arsenic-induced carcinogenesis and cancer stem cells

SEMINARS IN CANCER BIOLOGY

IF15.7

2021-11-0117

OAAI

Environmentally-induced mdig contributes to the severity of COVID-19 through fostering expression of SARS-CoV-2 receptor NRPs and glycan metabolism

THERANOSTICS

IF13.3

2021-01-019

OAAI

Mdig promotes oncogenic gene expression through antagonizing repressive histone methylation markers

THERANOSTICS

IF13.3

2020-01-0132

OAAI

Chitra Thakur

个人简介

教育背景

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