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Ziad Mallat

University of Cambridge
H指数:96
被引数:3.4万

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Anti-inflammatory therapy with low-dose IL-2 in acute coronary syndromes: a randomized phase 2 trial

急性冠状动脉综合征的低剂量IL-2抗炎治疗: 一项随机2期试验

2026-01-080
OAAI
Author Correction: Neuroimmune cardiovascular interfaces control atherosclerosis

作者更正: 神经免疫心血管接口控制动脉粥样硬化

Nature
IF48.5
2025-12-170
OAAI
AS-0098 Intermittent Statin Treatment Induces Innate Immune Reprogramming

AS-0098 间歇性他汀治疗诱导固有免疫重编程

JACC
IF0
2025-11-190
PRE
The essential role of sex and gender in atherosclerosis research: A statement from the Editorial Board of Atherosclerosis and Executive Committee of the European Atherosclerosis Society

动脉粥样硬化研究中性别(sex)与性别认同(gender)的核心作用:动脉粥样硬化杂志编辑委员会与欧洲动脉粥样硬化学会执行委员会的声明

2025-06-120
PRE
Low-Dose IL-2 Lowers Arterial Inflammation and Trends Towards Lower MACE in patients with ACS: The results of the IVORY trial and IVORY FINALE study

低剂量IL-2降低动脉炎症并呈现降低ACS患者主要不良心血管事件(MACE)的趋势:IVORY试验和IVORY FINALE研究的成果

2024-11-120
PRE
Early intermittent hyperlipidaemia alters tissue macrophages to fuel atherosclerosis
Nature
IF48.5
2024-09-040
OAAI
PLK1 inhibition dampens NLRP3 inflammasome-elicited response in inflammatory disease models

PLK1抑制抑制炎症性疾病模型中NLRP3炎性体引起的反应

2023-11-012
OAAI
Cell-autonomous regulation of complement C3 by factor H limits macrophage efferocytosis and exacerbates atherosclerosis

因子H对补体C3的细胞自主调节限制了巨噬细胞的分泌并加剧了动脉粥样硬化

IMMUNITY
IF26.3
2023-08-0119
OAAI
Genetic inhibition of CARD9 accelerates the development of atherosclerosis in mice through CD36 dependent-defective autophagy

CARD9基因抑制通过CD36依赖的自噬缺陷加速小鼠动脉粥样硬化的发展

2023-08-0111
OAAI