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COPIng with senescence
DOI:10.1038/s41556-023-01276-9.png)
摘要
En 中文
There is increasing interest in approaches that target and eliminate senescent cells. A study reports that the coatomer complex I (COPI) pathway is important for the survival of senescent cells, and suggests that targeting this pathway could hold therapeutic promise in the context of senescence-associated diseases.
期刊
IF:
19.1
论文数:
6.2K
被引数:
4.8W
机构
引用论文
Cellular senescence drives age-dependent hepatic steatosis细胞衰老驱动年龄依赖性肝脂肪变性
NATURE COMMUNICATIONS
IF15.7
COPI vesicle formation and N-myristoylation are targetable vulnerabilities of senescent cellsCOPI囊泡形成和N-肉豆蔻酰化是衰老细胞的可靶向脆弱性
NATURE CELL BIOLOGY
IF19.1
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