平艳艳 (Yanyan Ping)

Harbin Medical University教授邮箱:pingyanyan@hrbmu.edu.cn

H指数:18

被引数:2,815

个人简介

任职于生物信息科学与技术学院，教授，博士研究生学历。主要研究方向为肿瘤生物信息学。

教育背景

2008.09 – 2014.06
哈尔滨医科大学博士生物医学工程
2003.09 – 2008.06
哈尔滨医科大学学士生物技术

工作经历

2021.09 – 至今
哈尔滨医科大学,生物信息科学与技术学院
教授
2016.07 – 2021.09
哈尔滨医科大学,生物信息科学与技术学院
副教授
2016.05 – 2020.08
哈尔滨医科大学附属第三医院
博士后
2014.07 – 2016.09
哈尔滨医科大学,生物信息科学与技术学院
讲师

荣誉奖项

癌风险miRNA及协同调控风险通路（pathway）识别（2015）｜中华医学会
基于靶基因的逆向激活、共功能与高度互连识别胶质母细胞瘤中功能失调的miRNA-mRNA的调控模块（2014）｜哈尔滨市

Identifying Epithelial-Cell Progression Signatures (ECPSs) from Multi-Resolution Multi-Omics Data for Translational Clinical Applications in Lung Adenocarcinoma

从多分辨率多组学数据中识别上皮细胞进展特征（ECPSs），以用于肺腺癌的转化临床应用

International Journal of Molecular Sciences

IF4.9

2026-07-300

IMOP-Cancer: identifying mutation order pairs impacting cancer phenotypes

IMOP-Cancer：识别影响癌症表型的突变顺序对

BRIEFINGS IN BIOINFORMATICS

IF7.7

2025-07-010

PRE

Identifying the driver miRNAs with somatic copy number alterations driving dysregulated ceRNA networks in cancers

鉴定驱动癌症中ceRNA网络失调的体细胞拷贝数改变的驱动mirna

BIOLOGY DIRECT

IF4.9

2023-11-221

OAAI

Identifying the personalized driver gene sets maximally contributing to abnormality of transcriptome phenotype in glioblastoma multiforme individuals

MOLECULAR ONCOLOGY

IF4.5

2023-08-080

The Heterogeneous Cellular States of Glioblastoma Stem Cells Revealed by Single Cell Analysis

Stem Cells

IF3.6

2022-12-303

Revealing the functions of clonal driver gene mutations in patients based on evolutionary dependencies

基于进化依赖性揭示患者克隆驱动基因突变的功能

HUMAN MUTATION

IF3.7

2022-10-190

Systematic investigation of the prognostic impact of clonal status of somatic mutations across multiple cancer types

GENOMICS

IF3

2022-07-010

OAAI

Functional regulations between genetic alteration-driven genes and drug target genes acting as prognostic biomarkers in breast cancer

SCIENTIFIC REPORTS

IF3.9

2022-06-2311

OAAI

Revealing the contribution of somatic gene mutations to shaping tumor immune microenvironment

BRIEFINGS IN BIOINFORMATICS

IF7.7

2022-03-016

PRE

Dynamic regulatory networks of T cell trajectory dissect transcriptional control of T cell state transition

MOLECULAR THERAPY NUCLEIC ACIDS

IF6.1

2021-12-0114

OAAI

Single-Cell Transcriptomic Analysis Reveals a Tumor-Reactive T Cell Signature Associated With Clinical Outcome and Immunotherapy Response In Melanoma

单细胞转录组分析揭示了与黑色素瘤临床结果和免疫治疗反应相关的肿瘤反应性T细胞特征

FRONTIERS IN IMMUNOLOGY

IF5.9

2021-11-0527

OAAI

Dissecting the Functional Mechanisms of Somatic Copy-Number Alterations Based on Dysregulated ceRNA Networks across Cancers

MOLECULAR THERAPY-NUCLEIC ACIDS

IF0

2020-09-0112

OAAI

CellMarker: a manually curated resource of cell markers in human and mouse

CellMarker: 人类和小鼠细胞标记的手动策划资源

NUCLEIC ACIDS RESEARCH

IF13.1

2018-10-05892

OAAI

Identifying mutual exclusivity across cancer genomes: computational approaches to discover genetic interaction and reveal tumor vulnerability

BRIEFINGS IN BIOINFORMATICS

IF7.7

2017-08-3131

PREAI

Oncogenic long noncoding RNA landscape in breast cancer

MOLECULAR CANCER

IF33.9

2017-07-24196

OAAI

Cooperative genomic alteration network reveals molecular classification across 12 major cancer types

NUCLEIC ACIDS RESEARCH

IF13.1

2016-11-2922

OAAI

A comprehensive overview of lncRNA annotation resources

lncRNA注释资源的全面概述

BRIEFINGS IN BIOINFORMATICS

IF7.7

2016-03-04140

Chromatin states modify network motifs contributing to cell-specific functions

SCIENTIFIC REPORTS

IF3.9

2015-07-149

OAAI

Identifying core gene modules in glioblastoma based on multilayer factor-mediated dysfunctional regulatory networks through integrating multi-dimensional genomic data

NUCLEIC ACIDS RESEARCH

IF13.1

2015-02-0432

OAAI

Integrating epigenetic marks for identification of transcriptionally active miRNAs

整合表观遗传标记以鉴定转录活性miRNAs

GENOMICS

IF3

2014-08-013

OAAI