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收藏Promoters of ASCL1-and NEUROD1-dependent genes are specific targets of lurbinectedin in SCLC cells
Costanzo, Federico; Martinez Diez, Marta; Santamaria Nunez, Gema; Ignacio Diaz-Hernandez, Juan; Genes Robles, Carlos Mario; Diez Perez, Javier; Compe, Emmanuel; Ricci, Romeo; Li, Tsai-Kun; Coin, Frederic; Martinez Leal, Juan Fernando; Maria Garrido-Martin, Eva; Egly, Jean Marc
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收藏CDK7 and MITF repress a transcription program involved in survival and drug tolerance in melanoma
Berico, Pietro; Cigrang, Max; Davidson, Guillaume; Braun, Cathy; Sandoz, Jeremy; Legras, Stephanie; Vokshi, Bujamin Hektor; Slovic, Nevena; Peyresaubes, Francois; Robles, Carlos Mario Gene; Egly, Jean-Marc; Compe, Emmanuel; Davidson, Irwin; Coin, Frederic
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收藏The Cockayne syndrome group A and B proteins are part of a ubiquitin-proteasome degradation complex regulating cell division
Paccosi, Elena; Costanzo, Federico; Costantino, Michele; Balzerano, Alessio; Monteonofrio, Laura; Soddu, Silvia; Prantera, Giorgio; Brancorsini, Stefano; Egly, Jean-Marc; Proietti-De-Santis, Luca
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收藏The intricate network between the p34 and p44 subunits is central to the activity of the transcription/DNA repair factor TFIIH
Radu, Laura; Schoenwetter, Elisabeth; Braun, Cathy; Marcoux, Julien; Koelmel, Wolfgang; Schmitt, Dominik R.; Kuper, Jochen; Cianferani, Sarah; Egly, Jean M.; Poterszman, Arnaud; Kisker, Caroline
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收藏TFIIH-dependent MMP-1 overexpression in trichothiodystrophy leads to extracellular matrix alterations in patient skin
Arseni, Lavinia; Lanzafame, Manuela; Compe, Emmanuel; Fortugno, Paola; Afonso-Barroso, Antonio; Peverali, Fiorenzo A.; Lehmann, Alan R.; Zambruno, Giovanna; Egly, Jean-Marc; Stefanini, Miria; Orioli, Donata
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收藏Deletion of 5′ sequences of the CSB gene provides insight into the pathophysiology of Cockayne syndromeCSB基因5' 序列的缺失为Cockayne综合征的病理生理学提供了见识
Laugel, Vincent; Dalloz, Cecile; Stary, Anne; Cormier-Daire, Valerie; Desguerre, Isabelle; Renouil, Michel; Fourmaintraux, Alain; Velez-Cruz, Renier; Egly, Jean-Marc; Sarasin, Alain; Dollfus, Helene
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