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M

Manyun Dai

rutgers university system

H指数:15

被引数:821

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Persistent activation of growth hormone contributes to sexual dimorphic expression of hepatic drug metabolizing enzymes in Fgf15 transgenic mice

生长激素的持续激活有助于Fgf15转基因小鼠肝脏药物代谢酶的性二态性表达

Drug Metabolism and Disposition

IF4

2026-08-010

PRE

Endogenous phosphatidylcholine 16:0/20:4 increases to promote liver regeneration in hepatectomy

内源性磷脂酰胆碱16:0/20:4增加以促进肝切除术后的肝脏再生

IF4.1

2026-07-310

OAAI

Triptophenolide aggravates triptolide-induced liver injury through disrupting the biological function of intestine

Triptophenolide通过破坏肠道生物学功能加重triptolide诱导的肝脏损伤。

TOXICOLOGY AND APPLIED PHARMACOLOGY

IF3.4

2025-07-010

PRE

Gut-vascular barrier mediated Tripterygium wilfordii-induced liver injury: the synergism between triptolide and celastrol via intestinal FXR-ET-1 pathway

肠道血管屏障介导的雷公藤多苷诱导的肝损伤：雷公藤甲素和雷公藤红素通过肠道FXR-ET-1通路产生的协同作用

JOURNAL OF ETHNOPHARMACOLOGY

IF5.4

2025-06-010

PRE

Oxidative balance score and the potential for suffering rheumatoid arthritis: a cross-sectional study

FRONTIERS IN IMMUNOLOGY

IF5.9

2024-11-012

OAAI

A CDAHFD-induced mouse model mimicking human NASH in the metabolism of hepatic phosphatidylcholines and acyl carnitines

FOOD & FUNCTION

IF5.4

2024-01-013

PRE

Celastrol as an intestinal FXR inhibitor triggers tripolide-induced intestinal bleeding: Underlying mechanism of gastrointestinal injury induced by Tripterygium wilfordii

IF8.3

2023-12-014

PRE

Metabolomics Reveals Gut Microbiota Contribute to PPARα Deficiency-Induced Alcoholic Liver Injury

JOURNAL OF PROTEOME RESEARCH

IF3.6

2023-05-2613

PRE

Comparative Metabolomic Profiling of the Metabolic Differences of Δ9-Tetrahydrocannabinol and Cannabidiol

IF4.6

2022-11-045

OAAI

Polyamine metabolism links gut microbiota and testicular dysfunction

多胺代谢将肠道菌群与睾丸功能障碍联系起来

IF12.7

2021-11-1156

OAAI

Discovery of quality markers in Rubus Chingii Hu using UPLC-ESI-QTOF-MS

JOURNAL OF PHARMACEUTICAL AND BIOMEDICAL ANALYSIS

IF3.1

2021-09-0113

PREAI

Inhibition of inflammation by SP600125 in cholestatic liver injury is dependent on the administration-based exposure profile

INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE

IF5.8

2020-09-293

OA

Cholestatic models induced by lithocholic acid and α-naphthylisothiocyanate: Different etiological mechanisms for liver injury but shared JNK/STAT3 signaling

MOLECULAR MEDICINE REPORTS

IF3.5

2020-06-0315

OA

Gut microbiota protects from triptolide-induced hepatotoxicity: Key role of propionate and its downstream signalling events

PHARMACOLOGICAL RESEARCH

IF10.5

2020-05-0145

PREAI

Therapeutic action against chronic cholestatic liver injury by low-dose fenofibrate involves anti-chemotaxis via JNK-AP1-CCL2/CXCL2 signaling

PHARMACOLOGICAL REPORTS

IF3.8

2020-01-0810

PREAI

Metabolic Activation of Elemicin Leads to the Inhibition of Stearoyl-CoA Desaturase 1

CHEMICAL RESEARCH IN TOXICOLOGY

IF3.8

2019-08-306

OAAI

Targeted Metabolomics Reveals a Protective Role for Basal PPARα in Cholestasis Induced by α-Naphthylisothiocyanate

JOURNAL OF PROTEOME RESEARCH

IF3.6

2018-03-0224

OAAI

Inhibition of JNK signalling mediates PPAR-dependent protection against intrahepatic cholestasis by fenofibrate

BRITISH JOURNAL OF PHARMACOLOGY

IF7.7

2017-08-1046

OAAI

Oral administration of nano-titanium dioxide particle disrupts hepatic metabolic functions in a mouse model

ENVIRONMENTAL TOXICOLOGY AND PHARMACOLOGY

IF4.2

2017-01-0140

PREAI

Dual action of peroxisome proliferator-activated receptor alpha in perfluorodecanoic acid-induced hepatotoxicity

ARCHIVES OF TOXICOLOGY

IF6.9

2016-06-2522

OAAI