未登录

T

Teresa Priego

facultad de enfermeria

H指数:30

被引数:2,719

个人简介

暂无内容

教育背景

暂无内容

工作经历

暂无内容

荣誉奖项

暂无内容

Correction to: Relationship between muscle FNDC5, FGF21 mRNA, miR-150 and bone loss after sciatic denervation

更正：腓总神经去神经支配后肌肉FNDC5、FGF21 mRNA、miR-150与骨丢失之间的关系

Journal of Physiology and Biochemistry

IF4.3

2026-10-050

PRE

Relationship between muscle FNDC5, FGF21 mRNA, miR-150 and bone loss after sciatic denervation

腓总神经去神经支配后肌肉FNDC5、FGF21 mRNA、miR-150与骨量丢失的关系

Journal of Physiology and Biochemistry

IF4.3

2026-07-040

PRE

A Nutraceutical Product Based on a Mixture of Algae and Extra Virgin Olive Oils and Olive Leaf Extract Attenuates Sepsis-Induced Cardiovascular and Muscle Alterations in Rats

FRONTIERS IN NUTRITION

IF5.1

2022-06-204

OAAI

Addition of Olive Leaf Extract to a Mixture of Algae and Extra Virgin Olive Oils Decreases Fatty Acid Oxidation and Synergically Attenuates Age-Induced Hypertension, Sarcopenia and Insulin Resistance in Rats

IF6.6

2021-07-0115

OAAI

Olive Leaf Extract Supplementation to Old Wistar Rats Attenuates Aging-Induced Sarcopenia and Increases Insulin Sensitivity in Adipose Tissue and Skeletal Muscle

IF6.6

2021-05-0716

OAAI

Olive leaf extract supplementation improves the vascular and metabolic alterations associated with aging in Wistar rats

SCIENTIFIC REPORTS

IF3.9

2021-04-1416

OAAI

Beneficial Effects of a Mixture of Algae and Extra Virgin Olive Oils on the Age-Induced Alterations of Rodent Skeletal Muscle: Role of HDAC-4

IF5

2020-12-2511

OAAI

Identification of blood cell transcriptome-based biomarkers in adulthood predictive of increased risk to develop metabolic disorders using early life intervention rat models

IF4.2

2020-05-3111

PREAI

Peripheral Blood Cells, a Transcriptomic Tool in Nutrigenomic and Obesity Studies: Current State of the Art

COMPREHENSIVE REVIEWS IN FOOD SCIENCE AND FOOD SAFETY

IF14.1

2018-05-3028

OAAI

Sexual Dimorphism in the Age-Induced Insulin Resistance, Liver Steatosis, and Adipose Tissue Function in Rats

FRONTIERS IN PHYSIOLOGY

IF3.4

2017-07-1121

OAAI

TAS1R3 and UCN2 Transcript Levels in Blood Cells Are Associated With Sugary and Fatty Food Consumption in Children

JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM

IF5.1

2015-09-0113

OAAI

Maternal Fat Supplementation during Late Pregnancy and Lactation Influences the Development of Hepatic Steatosis in Offspring Depending on the Fat Source

JOURNAL OF AGRICULTURAL AND FOOD CHEMISTRY

IF6.2

2014-02-0614

PREAI

Potential interest of InsR, CPT1A, SLC27A2, FASN and PPARα expression in blood cells as biomarkers of dyslipidemia in children

PROCEEDINGS OF THE NUTRITION SOCIETY

IF4.5

2013-04-190

OAAI

Impaired insulin and leptin sensitivity in the offspring of moderate caloric-restricted dams during gestation is early programmed

JOURNAL OF NUTRITIONAL BIOCHEMISTRY

IF4.9

2012-12-0151

PREAI

Sex-differential expression of metabolism-related genes in response to a high-fat diet

IF4.7

2012-09-06106

OAAI

Metabolic programming of sirtuin 1 (SIRT1) expression by moderate energy restriction during gestation in rats may be related to obesity susceptibility in later life

BRITISH JOURNAL OF NUTRITION

IF3

2012-05-285

OAAI

Blood Cells as a Source of Transcriptional Biomarkers of Childhood Obesity and Its Related Metabolic Alterations: Results of the IDEFICS Study

JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM

IF5.1

2012-04-0144

OAAI

Metabolic programming of obesity by energy restriction during the perinatal period: different outcomes depending on gender and period, type and severity of restriction

FRONTIERS IN PHYSIOLOGY

IF3.4

2012-01-0172

OAAI

Moderate caloric restriction in lactating rats programs their offspring for a better response to HF diet feeding in a sex-dependent manner

JOURNAL OF NUTRITIONAL BIOCHEMISTRY

IF4.9

2011-06-0137

PREAI

Protective effects of leptin during the suckling period against later obesity may be associated with changes in promoter methylation of the hypothalamic pro-opiomelanocortin gene

BRITISH JOURNAL OF NUTRITION

IF3

2011-05-0455

OAAI