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Bruce L. Miller

University of California San Francisco
H指数:201
被引数:18.9万

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Speech clocks decode dementia phenotypes, social exposome, and biological aging

语音时钟解码痴呆表型、社会暴露组与生物衰老

2026-09-300
OA
Agentic AI for scaling diagnosis and care in neurodegenerative disease

代理式人工智能用于扩展神经退行性疾病的诊断与护理

2026-07-300
PRE
Clinical recognition of frontotemporal dementia with right temporal predominance: a consensus statement from the International Working Group

右颞叶为主的前额叶痴呆的临床识别:国际工作组共识声明

2025-12-120
OAAI
Cardiovascular risk factors and the allostatic interoceptive network in dementia

心血管风险因素及阿尔茨海默病的代偿性内感受网络

2025-11-220
OA
The Neighborhoods Study: Examining the social exposome in Alzheimer's disease and related dementias

邻里研究:考察阿尔茨海默病及相关痴呆症的社会暴露组

2025-11-130
OAAI
Blood-based Alzheimer's disease biomarkers in a behavioral neurology clinic: Real-world implementation, clinical utility, and diagnostic performance

基于血液的阿尔茨海默病生物标志物在行为神经病学诊所的应用:真实世界实施、临床效用和诊断性能

2025-11-130
OAAI
Combining plasma neurofilament light chain and frontotemporal atrophy improves differentiation of bvFTD from primary psychiatric disorders

结合血浆神经丝蛋白轻链和额颞叶萎缩,可提高对bvFTD与原发性精神障碍的鉴别诊断能力。

2025-10-080
OAAI
A single-cell, long-read, isoform-resolved case-control study of FTD reveals cell-type-specific and broad splicing dysregulation in human brain

一项单细胞、长读长、异构体解析的FTD病例对照研究揭示了人类大脑中细胞类型特异性和广泛的剪接失调。

2025-09-050
OAAI
β-Amyloid induces microglial expression of GPC4 and APOE leading to increased neuronal tau pathology and toxicity

β-amyloid诱导小胶质细胞表达GPC4和APOE,导致神经元tau病理和毒性增加。

2025-09-010
OAAI
Distinct manifestations of excitatory-inhibitory imbalance associated with amyloid-β and tau in patients with Alzheimer’s disease

与阿尔茨海默病患者中淀粉样蛋白-β和tau相关的兴奋-抑制失衡的不同表现

2025-08-260
OAAI
Interpersonal Physiological Linkage Between People With Behavioral Variant Frontotemporal Dementia and Alzheimer's Disease and Their Informal Caregivers

行为变异性额颞叶痴呆与阿尔茨海默病患者及其非正式照护者之间的人际生理关联

2025-08-060
OAAI