arrow
M

Manolis Pasparakis

University of Cologne
H指数:98
被引数:4.4万

个人简介

暂无内容

教育背景

暂无内容

工作经历

暂无内容

荣誉奖项

暂无内容

Cytotoxic CD4+ T cells induce age-associated myelopoiesis through CCL5–CCR5 signaling

细胞毒性CD4+ T细胞通过CCL5–CCR5信号通路诱导年龄相关性髓系增生

2026-08-260
OA
Oncogenic KRAS-driven type I interferon signalling primes pancreatic cancer for necroptosis

KRAS致癌基因驱动的I型干扰素信号为胰腺癌的坏死性凋亡奠定基础

2026-06-150
OAAI
Loss of MLKL impairs abdominal aortic aneurysm development by attenuating smooth muscle cell necroptosis

MLKL缺失通过减弱平滑肌细胞坏死性凋亡来抑制腹主动脉瘤的发展

2026-02-110
OAAI
DNA-protein cross-links promote cGAS-STING–driven premature aging and embryonic lethality

DNA-蛋白质交联促进cGAS-STING驱动的过早衰老和胚胎致死率

Science
IF45.8
2026-01-290
PRE
Lack of caspase 8 directs neuronal progenitor-like reprogramming and small cell lung cancer progression

缺乏caspase 8导致神经祖细胞样重编程和小细胞肺癌进展

2025-12-180
OAAI
Low Caspase 8 Expression Promotes Neuronal Progenitor Mimicry and Small Cell Lung Cancer Metastasis

低Caspase 8表达促进神经元祖细胞拟态和小细胞肺癌转移

2025-08-290
PRE
Endothelial CCR2 Signaling Induced by Colon Carcinoma Cells Enables Extravasation via the JAK2-Stat5 and p38MAPK Pathway (vol 22, pg 91, 2012)

由结肠癌细胞诱导的内皮CCR2信号通过JAK2-Stat5和p38MAPK通路促进外渗 (vol 22, pg 91, 2012)

2025-05-010
PRE
MLKL activity requires a splicing-regulated, druggable intramolecular interaction

MLKL的活性需要一种受剪接调控的、可成药的分子内相互作用。

2025-04-010
OAAI
Incontinentia pigmenti underlies thymic dysplasia, autoantibodies to type I IFNs, and viral diseases

色素失禁是胸腺发育不良,I型ifn自身抗体和病毒性疾病的基础

2024-10-011
OAAI