Sandra J. Friezner Degen

机构信息待补充

23H指数

60论文数

2.1K被引数

收录论文 14

发表时间

Rescue of prothrombin-deficiency by transgene expression in mice

THROMBOSIS AND HAEMOSTASIS

IF4.3

2017-12-09

PREAI

Sun, WY; Coleman, MJ; Witte, DP; Degen, SJF

Short-form Ron receptor is required for normal IFN-γ production in concanavalin A-induced acute liver injury

AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY

IF3.3

2007-01-01

PREAI

Wetzel, Cynthia C.; Leonis, Mike A.; Dent, Arlene; Olson, Meredith A.; Longmeier, Angela M.; Ney, Paul A.; Boivin, Greg P.; Kader, Sarah A.; Caldwell, Charles C.; Degen, Sandra J. F.; Waltz, Susan E.

Pediatric faculty members' attitudes about part-time faculty positions and policies to support part-time faculty: A study at one medical center

ACADEMIC MEDICINE

IF5.2

2005-10-01

OAAI

Kahn, JA; Degen, SJF; Mansour, ME; Goodman, E; Zeller, MH; Laor, T; Lanphear, NE; Boat, TF

Cross-talk between the receptor tyrosine kinases Ron and epidermal growth factor receptor

EXPERIMENTAL CELL RESEARCH

IF3.5

2003-10-01

PREAI

Peace, BE; Hill, KJ; Degen, SJF; Waltz, SE

Receptor tyrosine kinase ron is expressed in mouse reproductive tissues during embryo implantation and is important in trophoblast cell function

BIOLOGY OF REPRODUCTION

IF3

2003-04-01

OAAI

Hess, KA; Waltz, SE; Chan, EL; Degen, SJF

Deletion of the Ron receptor tyrosine kinase domain in mice provides protection from endotoxin-induced acute liver failure

HEPATOLOGY

IF15.8

2002-11-01

OAAI

Leonis, NA; Toney-Earley, K; Degen, SJF; Waltz, SE

The role of the receptor tyrosine kinase Ron in nickel-induced acute lung injury

AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY

IF5.3

2002-01-01

PREAI

McDowell, SA; Mallakin, A; Bachurski, CJ; Toney-Earley, K; Prows, DR; Bruno, T; Kaestner, KH; Witte, DP; Melin-Aldana, H; Degen, SJF; Leikauf, GD; Waltz, SE

Point mutations and overexpression of Ron induce transformation, tumor formation, and metastasis

ONCOGENE

IF7.3

2001-10-12

PREAI

Peace, BE; Hughes, MJ; Degen, SJF; Waltz, SE

Ron-mediated cytoplasmic signaling is dispensable for viability but is required to limit inflammatory responses

JOURNAL OF CLINICAL INVESTIGATION

IF13.6

2001-08-15

115

OAAI