S
Simon Haas
H指数:33
被引数:8,302
浏览量:0
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Author Correction: A kinetics-based model of haematopoiesis reveals extrinsic regulation of skewed lineage output from stem cells
作者更正:基于造血动力学的模型揭示了干细胞倾斜谱系输出受外源性调节
Nature Cell Biology
IF19.1
A distinct CAR-T cell phenotype mediates therapeutic response at limited doses
一种独特的CAR-T细胞表型介导了有限剂量下的治疗效果
Nature Communications
IF15.7
Oncogene inactivation-induced senescence facilitates tumor relapse
癌基因失活诱导的衰老促进肿瘤复发
Nature Communications
IF15.7
A kinetics-based model of haematopoiesis reveals extrinsic regulation of skewed lineage output from stem cells
基于造血动力学的模型揭示了干细胞倾斜谱系输出受外在调节
Nature Cell Biology
IF19.1
Natural killer cell immunotherapy reverses lung fibrosis by eliminating senescent fibroblasts
自然杀伤细胞免疫疗法通过清除衰老成纤维细胞逆转肺纤维化
Reprogramming of stroma-derived chemokine networks drives the loss of tissue organization in nodal B cell lymphoma
基质衍生的趋化因子网络的重编程驱动结节b细胞淋巴瘤中组织组织的丧失
Nature Cancer
IF28.5
Investigation of a global mouse methylome atlas reveals subtype-specific copy number alterations in pediatric cancer models
全球小鼠甲基化图谱的研究揭示了小儿癌症模型中的亚型特异性拷贝数改变
Nature Genetics
IF29
Bispecific BAFF-R/BCMA CAR T cells control growth of heterogeneous plasma cells in multiple myeloma
双特异性BAFF-R/BCMA CAR T细胞控制多发性骨髓瘤中异质性浆细胞的生长
Chronic inflammatory demyelinating polyneuropathy (CIDP) after cilta-cel therapy
接受cilta-cel治疗后发生的慢性炎症性脱髓鞘性多发性神经病 (CIDP)
Blood Cancer Journal
IF11.6
Ultra-high-scale cytometry-based cellular interaction mapping
基于细胞计数的超高尺度细胞相互作用图谱
Nature Methods
IF32.1
Aggressive Lymphoma after CD19 CAR T-Cell Therapy
CD19 CAR T细胞治疗后侵袭性淋巴瘤
Proteomic signatures improve risk prediction for common and rare diseases
蛋白质组学特征改善了常见和罕见疾病的风险预测
NATURE MEDICINE
IF50

